|
تضامنًا مع حق الشعب الفلسطيني |
JUNB
An Error has occured retrieving Wikidata item for infobox JUNB (JunB proto-oncogene, AP-1 transcription factor subunit) هوَ بروتين يُشَفر بواسطة جين JUNB في الإنسان.[1][2]
الوظيفة
![[أيقونة]](/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ Schütte J، Viallet J، Nau M، Segal S، Fedorko J، Minna J (فبراير 1990). "jun-B inhibits and c-fos stimulates the transforming and trans-activating activities of c-jun". Cell. ج. 59 ع. 6: 987–997. DOI:10.1016/0092-8674(89)90755-1. PMID:2513129.
- ^ "Entrez Gene: JUNB jun B proto-oncogene". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Hsu JC، Bravo R، Taub R (1992). "Interactions among LRF-1, JunB, c-Jun, and c-Fos define a regulatory program in the G1 phase of liver regeneration". Mol. Cell. Biol. ج. 12 ع. 10: 4654–65. PMC:360392. PMID:1406655.
- Zafarullah M، Martel-Pelletier J، Cloutier JM، Gedamu L، Pelletier JP (1992). "Expression of c-fos, c-jun, jun-B, metallothionein and metalloproteinase genes in human chondrocyte". FEBS Lett. ج. 306 ع. 2–3: 169–172. DOI:10.1016/0014-5793(92)80992-P. PMID:1633872.
- Mollinedo F، Vaquerizo MJ، Naranjo JR (1991). "Expression of c-jun, jun B and jun D proto-oncogenes in human peripheral-blood granulocytes". Biochem. J. 273(Pt 2): 477–9. PMC:1149869. PMID:1899335.
- Nomura N، Ide M، Sasamoto S، Matsui M، Date T، Ishizaki R (1990). "Isolation of human cDNA clones of jun-related genes, jun-B and jun-D". Nucleic Acids Res. ج. 18 ع. 10: 3047–3048. DOI:10.1093/nar/18.10.3047. PMC:330838. PMID:2112242.
- Maruyama K، Sugano S (1994). "Oligo-capping: a simple method to replace the cap structure of eukaryotic mRNAs with oligoribonucleotides". Gene. ج. 138 ع. 1–2: 171–174. DOI:10.1016/0378-1119(94)90802-8. PMID:8125298.
- Trask B، Fertitta A، Christensen M، Youngblom J، Bergmann A، Copeland A، de Jong P، Mohrenweiser H، Olsen A، Carrano A (1993). "Fluorescence in situ hybridization mapping of human chromosome 19: cytogenetic band location of 540 cosmids and 70 genes or DNA markers". Genomics. ج. 15 ع. 1: 133–145. DOI:10.1006/geno.1993.1021. PMID:8432525.
- Phinney DG، Tseng SW، Ryder K (1996). "Complex genetic organization of junB: multiple blocks of flanking evolutionarily conserved sequence at the murine and human junB loci". Genomics. ج. 28 ع. 2: 228–234. DOI:10.1006/geno.1995.1135. PMID:8530030.
- Dorsey MJ، Tae HJ، Sollenberger KG، Mascarenhas NT، Johansen LM، Taparowsky EJ (1996). "B-ATF: a novel human bZIP protein that associates with members of the AP-1 transcription factor family". Oncogene. ج. 11 ع. 11: 2255–65. PMID:8570175.
- Neyns B، Vermeij J، Bourgain C، Vandamme B، Amfo K، Lissens W، DeSutter P، Hooghe-Peters E، DeGrève J (1996). "Expression of the jun family of genes in human ovarian cancer and normal ovarian surface epithelium". Oncogene. ج. 12 ع. 6: 1247–57. PMID:8649827.
- Mendelson KG، Contois LR، Tevosian SG، Davis RJ، Paulson KE (1996). "Independent regulation of JNK/p38 mitogen-activated protein kinases by metabolic oxidative stress in the liver". Proc. Natl. Acad. Sci. U.S.A. ج. 93 ع. 23: 12908–12913. DOI:10.1073/pnas.93.23.12908. PMC:24019. PMID:8917518.
- Aronheim A، Zandi E، Hennemann H، Elledge SJ، Karin M (1997). "Isolation of an AP-1 repressor by a novel method for detecting protein-protein interactions". Mol. Cell. Biol. ج. 17 ع. 6: 3094–102. DOI:10.1128/mcb.17.6.3094. PMC:232162. PMID:9154808.
- Suzuki Y، Yoshitomo-Nakagawa K، Maruyama K، Suyama A، Sugano S (1997). "Construction and characterization of a full length-enriched and a 5'-end-enriched cDNA library". Gene. ج. 200 ع. 1–2: 149–156. DOI:10.1016/S0378-1119(97)00411-3. PMID:9373149.
- Fuchs SY، Xie B، Adler V، Fried VA، Davis RJ، Ronai Z (1998). "c-Jun NH2-terminal kinases target the ubiquitination of their associated transcription factors". J. Biol. Chem. ج. 272 ع. 51: 32163–32168. DOI:10.1074/jbc.272.51.32163. PMID:9405416.
- Venugopal R، Jaiswal AK (1999). "Nrf2 and Nrf1 in association with Jun proteins regulate antioxidant response element-mediated expression and coordinated induction of genes encoding detoxifying enzymes". Oncogene. ج. 17 ع. 24: 3145–3156. DOI:10.1038/sj.onc.1202237. PMID:9872330.
- Li B، Tournier C، Davis RJ، Flavell RA (1999). "Regulation of IL-4 expression by the transcription factor JunB during T helper cell differentiation". EMBO J. ج. 18 ع. 2: 420–432. DOI:10.1093/emboj/18.2.420. PMC:1171136. PMID:9889198.
- Liberati NT، Datto MB، Frederick JP، Shen X، Wong C، Rougier-Chapman EM، Wang XF (1999). "Smads bind directly to the Jun family of AP-1 transcription factors". Proc. Natl. Acad. Sci. U.S.A. ج. 96 ع. 9: 4844–4849. DOI:10.1073/pnas.96.9.4844. PMC:21779. PMID:10220381.
- Chen P، Flory E، Avots A، Jordan BW، Kirchhoff F، Ludwig S، Rapp UR (2000). "Transactivation of naturally occurring HIV-1 long terminal repeats by the JNK signaling pathway. The most frequent naturally occurring length polymorphism sequence introduces a novel binding site for AP-1 factors". J. Biol. Chem. ج. 275 ع. 27: 20382–20390. DOI:10.1074/jbc.M001149200. PMID:10764760.
- Echlin DR، Tae HJ، Mitin N، Taparowsky EJ (2000). "B-ATF functions as a negative regulator of AP-1 mediated transcription and blocks cellular transformation by Ras and Fos". Oncogene. ج. 19 ع. 14: 1752–1763. DOI:10.1038/sj.onc.1203491. PMID:10777209.
- Verrecchia F، Pessah M، Atfi A، Mauviel A (2000). "Tumor necrosis factor-alpha inhibits transforming growth factor-beta /Smad signaling in human dermal fibroblasts via AP-1 activation". J. Biol. Chem. ج. 275 ع. 39: 30226–30231. DOI:10.1074/jbc.M005310200. PMID:10903323.
