LITAF
من أرابيكا، الموسوعة العربية الحرة
المزيد من اللغات
المزيد من الإجراءات
An Error has occured retrieving Wikidata item for infobox LITAF (Lipopolysaccharide induced TNF factor) هوَ بروتين يُشَفر بواسطة جين LITAF في الإنسان.[1][2][3]
الوظيفة
![[أيقونة]](/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ "Entrez Gene: LITAF lipopolysaccharide-induced TNF factor". مؤرشف من الأصل في 2010-03-07.
- ^ Myokai F، Takashiba S، Lebo R، Amar S (مايو 1999). "A novel lipopolysaccharide-induced transcription factor regulating tumor necrosis factor alpha gene expression: molecular cloning, sequencing, characterization, and chromosomal assignment". Proc Natl Acad Sci U S A. ج. 96 ع. 8: 4518–23. DOI:10.1073/pnas.96.8.4518. PMC:16364. PMID:10200294.
- ^ Polyak K، Xia Y، Zweier JL، Kinzler KW، Vogelstein B (سبتمبر 1997). "A model for p53-induced apoptosis". Nature. ج. 389 ع. 6648: 300–5. DOI:10.1038/38525. PMID:9305847.
قراءة متعمقة
- Jolliffe CN، Harvey KF، Haines BP، وآخرون (2001). "Identification of multiple proteins expressed in murine embryos as binding partners for the WW domains of the ubiquitin-protein ligase Nedd4". Biochem. J. ج. 351 ع. 3: 557–65. DOI:10.1042/0264-6021:3510557. PMC:1221394. PMID:11042109.
- Moriwaki Y، Begum NA، Kobayashi M، وآخرون (2001). "Mycobacterium bovis Bacillus Calmette-Guerin and its cell wall complex induce a novel lysosomal membrane protein, SIMPLE, that bridges the missing link between lipopolysaccharide and p53-inducible gene, LITAF(PIG7), and estrogen-inducible gene, EET-1". J. Biol. Chem. ج. 276 ع. 25: 23065–76. DOI:10.1074/jbc.M011660200. PMID:11274176.
- Street VA، Goldy JD، Golden AS، وآخرون (2002). "Mapping of Charcot-Marie-Tooth disease type 1C to chromosome 16p identifies a novel locus for demyelinating neuropathies". Am. J. Hum. Genet. ج. 70 ع. 1: 244–50. DOI:10.1086/337943. PMC:384893. PMID:11713717.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Street VA، Bennett CL، Goldy JD، وآخرون (2003). "Mutation of a putative protein degradation gene LITAF/SIMPLE in Charcot-Marie-Tooth disease 1C". Neurology. ج. 60 ع. 1: 22–6. DOI:10.1212/wnl.60.1.22. PMID:12525712.
- Matsuda A، Suzuki Y، Honda G، وآخرون (2003). "Large-scale identification and characterization of human genes that activate NF-kappaB and MAPK signaling pathways". Oncogene. ج. 22 ع. 21: 3307–18. DOI:10.1038/sj.onc.1206406. PMID:12761501.
- Street VA، Bennett CL، Bird TD، Chance PF (2004). "New gene for CMT". J. Peripher. Nerv. Syst. ج. 8 ع. 4: 206. DOI:10.1111/j.1085-9489.2003.03025.x. PMID:14641644.
- Ota T، Suzuki Y، Nishikawa T، وآخرون (2004). "Complete sequencing and characterization of 21,243 full-length human cDNAs". Nat. Genet. ج. 36 ع. 1: 40–5. DOI:10.1038/ng1285. PMID:14702039.
- Ludes-Meyers JH، Kil H، Bednarek AK، وآخرون (2004). "WWOX binds the specific proline-rich ligand PPXY: identification of candidate interacting proteins". Oncogene. ج. 23 ع. 29: 5049–55. DOI:10.1038/sj.onc.1207680. PMC:4143251. PMID:15064722.
- Bennett CL، Shirk AJ، Huynh HM، وآخرون (2004). "SIMPLE mutation in demyelinating neuropathy and distribution in sciatic nerve". Ann. Neurol. ج. 55 ع. 5: 713–20. DOI:10.1002/ana.20094. PMID:15122712.
- Matsumura Y، Matsumura Y، Nishigori C، وآخرون (2004). "PIG7/LITAF gene mutation and overexpression of its gene product in extramammary Paget's disease". Int. J. Cancer. ج. 111 ع. 2: 218–23. DOI:10.1002/ijc.20251. PMID:15197774.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Saifi GM، Szigeti K، Wiszniewski W، وآخرون (2006). "SIMPLE mutations in Charcot-Marie-Tooth disease and the potential role of its protein product in protein degradation". Hum. Mutat. ج. 25 ع. 4: 372–83. DOI:10.1002/humu.20153. PMID:15776429.
- Meggouh F، de Visser M، Arts WF، وآخرون (2005). "Early onset neuropathy in a compound form of Charcot-Marie-Tooth disease". Ann. Neurol. ج. 57 ع. 4: 589–91. DOI:10.1002/ana.20434. PMID:15786462.
- Tang X، Marciano DL، Leeman SE، Amar S (2005). "LPS induces the interaction of a transcription factor, LPS-induced TNF-alpha factor, and STAT6(B) with effects on multiple cytokines". Proc. Natl. Acad. Sci. U.S.A. ج. 102 ع. 14: 5132–7. DOI:10.1073/pnas.0501159102. PMC:555602. PMID:15793005.
- Beauvais K، Furby A، Latour P (2006). "Clinical, electrophysiological and molecular genetic studies in a family with X-linked dominant Charcot-Marie-Tooth neuropathy presenting a novel mutation in GJB1 Promoter and a rare polymorphism in LITAF/SIMPLE". Neuromuscul. Disord. ج. 16 ع. 1: 14–8. DOI:10.1016/j.nmd.2005.09.008. PMID:16373087.
- Latour P، Gonnaud PM، Ollagnon E، وآخرون (2006). "SIMPLE mutation analysis in dominant demyelinating Charcot-Marie-Tooth disease: three novel mutations". J. Peripher. Nerv. Syst. ج. 11 ع. 2: 148–55. DOI:10.1111/j.1085-9489.2006.00080.x. PMID:16787513.
- Stucchi A، Reed K، O'Brien M، وآخرون (2007). "A new transcription factor that regulates TNF-alpha gene expression, LITAF, is increased in intestinal tissues from patients with CD and UC". Inflamm. Bowel Dis. ج. 12 ع. 7: 581–7. DOI:10.1097/01.MIB.0000225338.14356.d5. PMID:16804395.
