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LITAF
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An Error has occured retrieving Wikidata item for infobox LITAF (Lipopolysaccharide induced TNF factor) هوَ بروتين يُشَفر بواسطة جين LITAF في الإنسان.[1][2][3]
الوظيفة
![[أيقونة]](/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ "Entrez Gene: LITAF lipopolysaccharide-induced TNF factor". مؤرشف من الأصل في 2010-03-07.
- ^ Myokai F، Takashiba S، Lebo R، Amar S (مايو 1999). "A novel lipopolysaccharide-induced transcription factor regulating tumor necrosis factor alpha gene expression: molecular cloning, sequencing, characterization, and chromosomal assignment". Proc Natl Acad Sci U S A. ج. 96 ع. 8: 4518–23. DOI:10.1073/pnas.96.8.4518. PMC:16364. PMID:10200294.
- ^ Polyak K، Xia Y، Zweier JL، Kinzler KW، Vogelstein B (سبتمبر 1997). "A model for p53-induced apoptosis". Nature. ج. 389 ع. 6648: 300–5. DOI:10.1038/38525. PMID:9305847.
قراءة متعمقة
- Jolliffe CN، Harvey KF، Haines BP، وآخرون (2001). "Identification of multiple proteins expressed in murine embryos as binding partners for the WW domains of the ubiquitin-protein ligase Nedd4". Biochem. J. ج. 351 ع. 3: 557–65. DOI:10.1042/0264-6021:3510557. PMC:1221394. PMID:11042109.
- Moriwaki Y، Begum NA، Kobayashi M، وآخرون (2001). "Mycobacterium bovis Bacillus Calmette-Guerin and its cell wall complex induce a novel lysosomal membrane protein, SIMPLE, that bridges the missing link between lipopolysaccharide and p53-inducible gene, LITAF(PIG7), and estrogen-inducible gene, EET-1". J. Biol. Chem. ج. 276 ع. 25: 23065–76. DOI:10.1074/jbc.M011660200. PMID:11274176.
- Street VA، Goldy JD، Golden AS، وآخرون (2002). "Mapping of Charcot-Marie-Tooth disease type 1C to chromosome 16p identifies a novel locus for demyelinating neuropathies". Am. J. Hum. Genet. ج. 70 ع. 1: 244–50. DOI:10.1086/337943. PMC:384893. PMID:11713717.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Street VA، Bennett CL، Goldy JD، وآخرون (2003). "Mutation of a putative protein degradation gene LITAF/SIMPLE in Charcot-Marie-Tooth disease 1C". Neurology. ج. 60 ع. 1: 22–6. DOI:10.1212/wnl.60.1.22. PMID:12525712.
- Matsuda A، Suzuki Y، Honda G، وآخرون (2003). "Large-scale identification and characterization of human genes that activate NF-kappaB and MAPK signaling pathways". Oncogene. ج. 22 ع. 21: 3307–18. DOI:10.1038/sj.onc.1206406. PMID:12761501.
- Street VA، Bennett CL، Bird TD، Chance PF (2004). "New gene for CMT". J. Peripher. Nerv. Syst. ج. 8 ع. 4: 206. DOI:10.1111/j.1085-9489.2003.03025.x. PMID:14641644.
- Ota T، Suzuki Y، Nishikawa T، وآخرون (2004). "Complete sequencing and characterization of 21,243 full-length human cDNAs". Nat. Genet. ج. 36 ع. 1: 40–5. DOI:10.1038/ng1285. PMID:14702039.
- Ludes-Meyers JH، Kil H، Bednarek AK، وآخرون (2004). "WWOX binds the specific proline-rich ligand PPXY: identification of candidate interacting proteins". Oncogene. ج. 23 ع. 29: 5049–55. DOI:10.1038/sj.onc.1207680. PMC:4143251. PMID:15064722.
- Bennett CL، Shirk AJ، Huynh HM، وآخرون (2004). "SIMPLE mutation in demyelinating neuropathy and distribution in sciatic nerve". Ann. Neurol. ج. 55 ع. 5: 713–20. DOI:10.1002/ana.20094. PMID:15122712.
- Matsumura Y، Matsumura Y، Nishigori C، وآخرون (2004). "PIG7/LITAF gene mutation and overexpression of its gene product in extramammary Paget's disease". Int. J. Cancer. ج. 111 ع. 2: 218–23. DOI:10.1002/ijc.20251. PMID:15197774.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Saifi GM، Szigeti K، Wiszniewski W، وآخرون (2006). "SIMPLE mutations in Charcot-Marie-Tooth disease and the potential role of its protein product in protein degradation". Hum. Mutat. ج. 25 ع. 4: 372–83. DOI:10.1002/humu.20153. PMID:15776429.
- Meggouh F، de Visser M، Arts WF، وآخرون (2005). "Early onset neuropathy in a compound form of Charcot-Marie-Tooth disease". Ann. Neurol. ج. 57 ع. 4: 589–91. DOI:10.1002/ana.20434. PMID:15786462.
- Tang X، Marciano DL، Leeman SE، Amar S (2005). "LPS induces the interaction of a transcription factor, LPS-induced TNF-alpha factor, and STAT6(B) with effects on multiple cytokines". Proc. Natl. Acad. Sci. U.S.A. ج. 102 ع. 14: 5132–7. DOI:10.1073/pnas.0501159102. PMC:555602. PMID:15793005.
- Beauvais K، Furby A، Latour P (2006). "Clinical, electrophysiological and molecular genetic studies in a family with X-linked dominant Charcot-Marie-Tooth neuropathy presenting a novel mutation in GJB1 Promoter and a rare polymorphism in LITAF/SIMPLE". Neuromuscul. Disord. ج. 16 ع. 1: 14–8. DOI:10.1016/j.nmd.2005.09.008. PMID:16373087.
- Latour P، Gonnaud PM، Ollagnon E، وآخرون (2006). "SIMPLE mutation analysis in dominant demyelinating Charcot-Marie-Tooth disease: three novel mutations". J. Peripher. Nerv. Syst. ج. 11 ع. 2: 148–55. DOI:10.1111/j.1085-9489.2006.00080.x. PMID:16787513.
- Stucchi A، Reed K، O'Brien M، وآخرون (2007). "A new transcription factor that regulates TNF-alpha gene expression, LITAF, is increased in intestinal tissues from patients with CD and UC". Inflamm. Bowel Dis. ج. 12 ع. 7: 581–7. DOI:10.1097/01.MIB.0000225338.14356.d5. PMID:16804395.
