FLII
من أرابيكا، الموسوعة العربية الحرة
المزيد من اللغات
المزيد من الإجراءات
An Error has occured retrieving Wikidata item for infobox FLII (FLII, actin remodeling protein) هوَ بروتين يُشَفر بواسطة جين FLII في الإنسان.[1][2][3]
الوظيفة
![[أيقونة]](/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ Chen KS، Gunaratne PH، Hoheisel JD، Young IG، Miklos GL، Greenberg F، Shaffer LG، Campbell HD، Lupski JR (فبراير 1995). "The human homologue of the Drosophila melanogaster flightless-I gene (flil) maps within the Smith-Magenis microdeletion critical region in 17p11.2". Am J Hum Genet. ج. 56 ع. 1: 175–82. PMC:1801336. PMID:7825574.
- ^ Ghoshdastider U, Popp D, Burtnick LD, Robinson RC (2013). "The expanding superfamily of gelsolin homology domain proteins". Cytoskeleton (Hoboken). ج. 70 ع. 11: 775–95. DOI:10.1002/cm.21149. PMID:24155256.
- ^ "Entrez Gene: FLII flightless I homolog (Drosophila)". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Campbell HD، Schimansky T، Claudianos C، وآخرون (1994). "The Drosophila melanogaster flightless-I gene involved in gastrulation and muscle degeneration encodes gelsolin-like and leucine-rich repeat domains and is conserved in Caenorhabditis elegans and humans". Proc. Natl. Acad. Sci. U.S.A. ج. 90 ع. 23: 11386–90. Bibcode:1993PNAS...9011386C. DOI:10.1073/pnas.90.23.11386. PMC:47987. PMID:8248259.
- Campbell HD، Fountain S، Young IG، وآخرون (1997). "Genomic structure, evolution, and expression of human FLII, a gelsolin and leucine-rich-repeat family member: overlap with LLGL". Genomics. ج. 42 ع. 1: 46–54. DOI:10.1006/geno.1997.4709. PMID:9177775.
- Liu YT، Yin HL (1998). "Identification of the binding partners for flightless I, A novel protein bridging the leucine-rich repeat and the gelsolin superfamilies". J. Biol. Chem. ج. 273 ع. 14: 7920–7. DOI:10.1074/jbc.273.14.7920. PMID:9525888.
- Wilson SA، Brown EC، Kingsman AJ، Kingsman SM (1998). "TRIP: a novel double stranded RNA binding protein which interacts with the leucine rich repeat of flightless I." Nucleic Acids Res. ج. 26 ع. 15: 3460–7. DOI:10.1093/nar/26.15.3460. PMC:147727. PMID:9671805.
- Fong KS، de Couet HG (1999). "Novel proteins interacting with the leucine-rich repeat domain of human flightless-I identified by the yeast two-hybrid system". Genomics. ج. 58 ع. 2: 146–57. DOI:10.1006/geno.1999.5817. PMID:10366446.
- Davy DA، Campbell HD، Fountain S، وآخرون (2001). "The flightless I protein colocalizes with actin- and microtubule-based structures in motile Swiss 3T3 fibroblasts: evidence for the involvement of PI 3-kinase and Ras-related small GTPases". J. Cell Sci. ج. 114 ع. Pt 3: 549–62. PMID:11171324.
- Campbell HD، Fountain S، McLennan IS، وآخرون (2002). "Fliih, a gelsolin-related cytoskeletal regulator essential for early mammalian embryonic development". Mol. Cell. Biol. ج. 22 ع. 10: 3518–26. DOI:10.1128/MCB.22.10.3518-3526.2002. PMC:133791. PMID:11971982.
- Bi W، Yan J، Stankiewicz P، وآخرون (2002). "Genes in a refined Smith-Magenis syndrome critical deletion interval on chromosome 17p11.2 and the syntenic region of the mouse". Genome Res. ج. 12 ع. 5: 713–28. DOI:10.1101/gr.73702. PMC:186594. PMID:11997338.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. Bibcode:2002PNAS...9916899M. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Gevaert K، Goethals M، Martens L، وآخرون (2004). "Exploring proteomes and analyzing protein processing by mass spectrometric identification of sorted N-terminal peptides". Nat. Biotechnol. ج. 21 ع. 5: 566–9. DOI:10.1038/nbt810. PMID:12665801.
- Lee YH، Campbell HD، Stallcup MR (2004). "Developmentally essential protein flightless I is a nuclear receptor coactivator with actin binding activity". Mol. Cell. Biol. ج. 24 ع. 5: 2103–17. DOI:10.1128/MCB.24.5.2103-2117.2004. PMC:350567. PMID:14966289.
- Beausoleil SA، Jedrychowski M، Schwartz D، وآخرون (2004). "Large-scale characterization of HeLa cell nuclear phosphoproteins". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 33: 12130–5. Bibcode:2004PNAS..10112130B. DOI:10.1073/pnas.0404720101. PMC:514446. PMID:15302935.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Wang T، Chuang TH، Ronni T، وآخرون (2006). "Flightless I homolog negatively modulates the TLR pathway". J. Immunol. ج. 176 ع. 3: 1355–62. DOI:10.4049/jimmunol.176.3.1355. PMID:16424162.
- Beausoleil SA، Villén J، Gerber SA، وآخرون (2006). "A probability-based approach for high-throughput protein phosphorylation analysis and site localization". Nat. Biotechnol. ج. 24 ع. 10: 1285–92. DOI:10.1038/nbt1240. PMID:16964243.
- Olsen JV، Blagoev B، Gnad F، وآخرون (2006). "Global, in vivo, and site-specific phosphorylation dynamics in signaling networks". Cell. ج. 127 ع. 3: 635–48. DOI:10.1016/j.cell.2006.09.026. PMID:17081983.
