CUGBP2
من أرابيكا، الموسوعة العربية الحرة
المزيد من اللغات
المزيد من الإجراءات
An Error has occured retrieving Wikidata item for infobox CUGBP2 (CUGBP Elav-like family member 2) هوَ بروتين يُشَفر بواسطة جين CUGBP2 في الإنسان.[1][1][2][3]
الوظيفة
![[أيقونة]](/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ أ ب "Entrez Gene: CELF2 CUGBP, Elav-like family member 2". مؤرشف من الأصل في 2010-12-05.
- ^ Lu X، Timchenko NA، Timchenko LT (مارس 1999). "Cardiac elav-type RNA-binding protein (ETR-3) binds to RNA CUG repeats expanded in myotonic dystrophy". Hum Mol Genet. ج. 8 ع. 1: 53–60. DOI:10.1093/hmg/8.1.53. PMID:9887331.
- ^ Hwang DM، Hwang WS، Liew CC (مارس 1995). "Single pass sequencing of a unidirectional human fetal heart cDNA library to discover novel genes of the cardiovascular system". J Mol Cell Cardiol. ج. 26 ع. 10: 1329–33. DOI:10.1006/jmcc.1994.1151. PMID:7869393.
قراءة متعمقة
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- Li D، Bachinski LL، Roberts R (2001). "Genomic organization and isoform-specific tissue expression of human NAPOR (CUGBP2) as a candidate gene for familial arrhythmogenic right ventricular dysplasia". Genomics. ج. 74 ع. 3: 396–401. DOI:10.1006/geno.2001.6558. PMID:11414768.
- Anant S، Henderson JO، Mukhopadhyay D، وآخرون (2002). "Novel role for RNA-binding protein CUGBP2 in mammalian RNA editing. CUGBP2 modulates C to U editing of apolipoprotein B mRNA by interacting with apobec-1 and ACF, the apobec-1 complementation factor". J. Biol. Chem. ج. 276 ع. 50: 47338–51. DOI:10.1074/jbc.M104911200. PMID:11577082.
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- Mukhopadhyay D، Houchen CW، Kennedy S، وآخرون (2003). "Coupled mRNA stabilization and translational silencing of cyclooxygenase-2 by a novel RNA binding protein, CUGBP2". Mol. Cell. ج. 11 ع. 1: 113–26. DOI:10.1016/S1097-2765(03)00012-1. PMID:12535526.
- Singh G، Charlet-B N، Han J، Cooper TA (2004). "ETR-3 and CELF4 protein domains required for RNA binding and splicing activity in vivo". Nucleic Acids Res. ج. 32 ع. 3: 1232–41. DOI:10.1093/nar/gkh275. PMC:373409. PMID:14973222.
- Mukhopadhyay D، Jung J، Murmu N، وآخرون (2004). "CUGBP2 plays a critical role in apoptosis of breast cancer cells in response to genotoxic injury". Ann. N. Y. Acad. Sci. ج. 1010: 504–9. Bibcode:2003NYASA1010..504M. DOI:10.1196/annals.1299.093. PMID:15033780.
- Brandenberger R، Wei H، Zhang S، وآخرون (2005). "Transcriptome characterization elucidates signaling networks that control human ES cell growth and differentiation". Nat. Biotechnol. ج. 22 ع. 6: 707–16. DOI:10.1038/nbt971. PMID:15146197.
- Suzuki Y، Yamashita R، Shirota M، وآخرون (2004). "Sequence Comparison of Human and Mouse Genes Reveals a Homologous Block Structure in the Promoter Regions". Genome Res. ج. 14 ع. 9: 1711–8. DOI:10.1101/gr.2435604. PMC:515316. PMID:15342556.
- Murmu N، Jung J، Mukhopadhyay D، وآخرون (2004). "Dynamic antagonism between RNA-binding protein CUGBP2 and cyclooxygenase-2-mediated prostaglandin E2 in radiation damage". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 38: 13873–8. Bibcode:2004PNAS..10113873M. DOI:10.1073/pnas.0406066101. PMC:518846. PMID:15358864.
- Han J، Cooper TA (2005). "Identification of CELF splicing activation and repression domains in vivo". Nucleic Acids Res. ج. 33 ع. 9: 2769–80. DOI:10.1093/nar/gki561. PMC:1126903. PMID:15894795.
- Kimura K، Wakamatsu A، Suzuki Y، وآخرون (2006). "Diversification of transcriptional modulation: Large-scale identification and characterization of putative alternative promoters of human genes". Genome Res. ج. 16 ع. 1: 55–65. DOI:10.1101/gr.4039406. PMC:1356129. PMID:16344560.
- Lim J، Hao T، Shaw C، وآخرون (2006). "A protein-protein interaction network for human inherited ataxias and disorders of Purkinje cell degeneration". Cell. ج. 125 ع. 4: 801–14. DOI:10.1016/j.cell.2006.03.032. PMID:16713569.
- Chen Z، Eggerman TL، Patterson AP (2007). "ApoB mRNA editing is mediated by a coordinated modulation of multiple apoB mRNA editing enzyme components". Am. J. Physiol. Gastrointest. Liver Physiol. ج. 292 ع. 1: G53–65. DOI:10.1152/ajpgi.00118.2006. PMID:16920700.
