تضامنًا مع حق الشعب الفلسطيني |
SH3GL2
اذهب إلى التنقل
اذهب إلى البحث
An Error has occured retrieving Wikidata item for infobox SH3GL2 (SH3 domain containing GRB2 like 2, endophilin A1) هوَ بروتين يُشَفر بواسطة جين SH3GL2 في الإنسان.[1][2]
الوظيفة
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ "Entrez Gene: SH3GL2 SH3-domain GRB2-like 2". مؤرشف من الأصل في 2010-12-05.
- ^ Giachino C؛ Lantelme E؛ Lanzetti L؛ Saccone S؛ Bella Valle G؛ Migone N (يوليو 1997). "A novel SH3-containing human gene family preferentially expressed in the central nervous system". Genomics. ج. 41 ع. 3: 427–34. DOI:10.1006/geno.1997.4645. PMID:9169142.
قراءة متعمقة
- Ren Y, Fang F, Davey F, Taylor M, Aiton J, Coote P, Yao J, Chen JX, Yan SD & Gunn-Moore FJ. (2008). "Endophilin I expression is increased in the brains of Alzheimer's disease patients". Journal of Biological Chemistry. ج. 283 ع. 9: 5685–91. DOI:10.1074/jbc.M707932200. PMID:18167351.
{{استشهاد بدورية محكمة}}
: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link) - Reutens AT؛ Begley CG (2002). "Endophilin-1: a multifunctional protein". Int. J. Biochem. Cell Biol. ج. 34 ع. 10: 1173–7. DOI:10.1016/S1357-2725(02)00063-8. PMID:12127567.
- So CW، Caldas C، Liu MM، وآخرون (1997). "EEN encodes for a member of a new family of proteins containing an Src homology 3 domain and is the third gene located on chromosome 19p13 that fuses to MLL in human leukemia". Proc. Natl. Acad. Sci. U.S.A. ج. 94 ع. 6: 2563–8. Bibcode:1997PNAS...94.2563S. DOI:10.1073/pnas.94.6.2563. PMC:20128. PMID:9122235.
- Ringstad N؛ Nemoto Y؛ De Camilli P (1997). "The SH3p4/Sh3p8/SH3p13 protein family: binding partners for synaptojanin and dynamin via a Grb2-like Src homology 3 domain". Proc. Natl. Acad. Sci. U.S.A. ج. 94 ع. 16: 8569–74. Bibcode:1997PNAS...94.8569R. DOI:10.1073/pnas.94.16.8569. PMC:23017. PMID:9238017.
- Micheva KD؛ Ramjaun AR؛ Kay BK؛ McPherson PS (1997). "SH3 domain-dependent interactions of endophilin with amphiphysin". FEBS Lett. ج. 414 ع. 2: 308–12. DOI:10.1016/S0014-5793(97)01016-8. PMID:9315708.
- Micheva KD؛ Kay BK؛ McPherson PS (1997). "Synaptojanin forms two separate complexes in the nerve terminal. Interactions with endophilin and amphiphysin". J. Biol. Chem. ج. 272 ع. 43: 27239–45. DOI:10.1074/jbc.272.43.27239. PMID:9341169.
- Howard L؛ Nelson KK؛ Maciewicz RA؛ Blobel CP (1999). "Interaction of the metalloprotease disintegrins MDC9 and MDC15 with two SH3 domain-containing proteins, endophilin I and SH3PX1". J. Biol. Chem. ج. 274 ع. 44: 31693–9. DOI:10.1074/jbc.274.44.31693. PMID:10531379.
- Tang Y، Hu LA، Miller WE، وآخرون (1999). "Identification of the endophilins (SH3p4/p8/p13) as novel binding partners for the beta1-adrenergic receptor". Proc. Natl. Acad. Sci. U.S.A. ج. 96 ع. 22: 12559–64. Bibcode:1999PNAS...9612559T. DOI:10.1073/pnas.96.22.12559. PMC:22990. PMID:10535961.
- Cestra G، Castagnoli L، Dente L، وآخرون (1999). "The SH3 domains of endophilin and amphiphysin bind to the proline-rich region of synaptojanin 1 at distinct sites that display an unconventional binding specificity". J. Biol. Chem. ج. 274 ع. 45: 32001–7. DOI:10.1074/jbc.274.45.32001. PMID:10542231.
- So CW، So CK، Cheung N، وآخرون (2000). "The interaction between EEN and Abi-1, two MLL fusion partners, and synaptojanin and dynamin: implications for leukaemogenesis". Leukemia. ج. 14 ع. 4: 594–601. DOI:10.1038/sj.leu.2401692. PMID:10764144.
- So CW، Sham MH، Chew SL، وآخرون (2000). "Expression and protein-binding studies of the EEN gene family, new interacting partners for dynamin, synaptojanin and huntingtin proteins". Biochem. J. ج. 348 ع. 2: 447–58. DOI:10.1042/0264-6021:3480447. PMC:1221085. PMID:10816441.
- Zucconi A، Dente L، Santonico E، وآخرون (2001). "Selection of ligands by panning of domain libraries displayed on phage lambda reveals new potential partners of synaptojanin 1". J. Mol. Biol. ج. 307 ع. 5: 1329–39. DOI:10.1006/jmbi.2001.4572. PMID:11292345.
- Ramjaun AR، Angers A، Legendre-Guillemin V، وآخرون (2001). "Endophilin regulates JNK activation through its interaction with the germinal center kinase-like kinase". J. Biol. Chem. ج. 276 ع. 31: 28913–9. DOI:10.1074/jbc.M103198200. PMID:11384986.
- Chen-Hwang MC؛ Chen HR؛ Elzinga M؛ Hwang YW (2002). "Dynamin is a minibrain kinase/dual specificity Yak1-related kinase 1A substrate". J. Biol. Chem. ج. 277 ع. 20: 17597–604. DOI:10.1074/jbc.M111101200. PMID:11877424.
- Soubeyran P، Kowanetz K، Szymkiewicz I، وآخرون (2002). "Cbl-CIN85-endophilin complex mediates ligand-induced downregulation of EGF receptors". Nature. ج. 416 ع. 6877: 183–7. Bibcode:2002Natur.416..183S. DOI:10.1038/416183a. PMID:11894095.
- Petrelli A، Gilestro GF، Lanzardo S، وآخرون (2002). "The endophilin-CIN85-Cbl complex mediates ligand-dependent downregulation of c-Met". Nature. ج. 416 ع. 6877: 187–90. Bibcode:2002Natur.416..187P. DOI:10.1038/416187a. PMID:11894096.
- Chatellard-Causse C، Blot B، Cristina N، وآخرون (2002). "Alix (ALG-2-interacting protein X), a protein involved in apoptosis, binds to endophilins and induces cytoplasmic vacuolization". J. Biol. Chem. ج. 277 ع. 32: 29108–15. DOI:10.1074/jbc.M204019200. PMID:12034747.
- Szymkiewicz I، Kowanetz K، Soubeyran P، وآخرون (2002). "CIN85 participates in Cbl-b-mediated down-regulation of receptor tyrosine kinases". J. Biol. Chem. ج. 277 ع. 42: 39666–72. DOI:10.1074/jbc.M205535200. PMID:12177062.
- Haglund K؛ Shimokawa N؛ Szymkiewicz I؛ Dikic I (2002). "Cbl-directed monoubiquitination of CIN85 is involved in regulation of ligand-induced degradation of EGF receptors". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 19: 12191–6. Bibcode:2002PNAS...9912191H. DOI:10.1073/pnas.192462299. PMC:129420. PMID:12218189.
- Verstreken P، Kjaerulff O، Lloyd TE، وآخرون (2002). "Endophilin mutations block clathrin-mediated endocytosis but not neurotransmitter release". Cell. ج. 109 ع. 1: 101–12. DOI:10.1016/S0092-8674(02)00688-8. PMID:11955450.