JUNB

An Error has occured retrieving Wikidata item for infobox JUNB‏ (JunB proto-oncogene, AP-1 transcription factor subunit) هوَ بروتين يُشَفر بواسطة جين JUNB في الإنسان.

قراءة متعمقة

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• Zafarullah M، Martel-Pelletier J، Cloutier JM، Gedamu L، Pelletier JP (1992). "Expression of c-fos, c-jun, jun-B, metallothionein and metalloproteinase genes in human chondrocyte". FEBS Lett. ج. 306 ع. 2–3: 169–172. DOI:10.1016/0014-5793(92)80992-P. PMID:1633872.
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• Phinney DG، Tseng SW، Ryder K (1996). "Complex genetic organization of junB: multiple blocks of flanking evolutionarily conserved sequence at the murine and human junB loci". Genomics. ج. 28 ع. 2: 228–234. DOI:10.1006/geno.1995.1135. PMID:8530030.
• Dorsey MJ، Tae HJ، Sollenberger KG، Mascarenhas NT، Johansen LM، Taparowsky EJ (1996). "B-ATF: a novel human bZIP protein that associates with members of the AP-1 transcription factor family". Oncogene. ج. 11 ع. 11: 2255–65. PMID:8570175.
• Neyns B، Vermeij J، Bourgain C، Vandamme B، Amfo K، Lissens W، DeSutter P، Hooghe-Peters E، DeGrève J (1996). "Expression of the jun family of genes in human ovarian cancer and normal ovarian surface epithelium". Oncogene. ج. 12 ع. 6: 1247–57. PMID:8649827.
• Mendelson KG، Contois LR، Tevosian SG، Davis RJ، Paulson KE (1996). "Independent regulation of JNK/p38 mitogen-activated protein kinases by metabolic oxidative stress in the liver". Proc. Natl. Acad. Sci. U.S.A. ج. 93 ع. 23: 12908–12913. DOI:10.1073/pnas.93.23.12908. PMC:24019. PMID:8917518.
• Aronheim A، Zandi E، Hennemann H، Elledge SJ، Karin M (1997). "Isolation of an AP-1 repressor by a novel method for detecting protein-protein interactions". Mol. Cell. Biol. ج. 17 ع. 6: 3094–102. DOI:10.1128/mcb.17.6.3094. PMC:232162. PMID:9154808.
• Suzuki Y، Yoshitomo-Nakagawa K، Maruyama K، Suyama A، Sugano S (1997). "Construction and characterization of a full length-enriched and a 5'-end-enriched cDNA library". Gene. ج. 200 ع. 1–2: 149–156. DOI:10.1016/S0378-1119(97)00411-3. PMID:9373149.
• Fuchs SY، Xie B، Adler V، Fried VA، Davis RJ، Ronai Z (1998). "c-Jun NH2-terminal kinases target the ubiquitination of their associated transcription factors". J. Biol. Chem. ج. 272 ع. 51: 32163–32168. DOI:10.1074/jbc.272.51.32163. PMID:9405416.
• Venugopal R، Jaiswal AK (1999). "Nrf2 and Nrf1 in association with Jun proteins regulate antioxidant response element-mediated expression and coordinated induction of genes encoding detoxifying enzymes". Oncogene. ج. 17 ع. 24: 3145–3156. DOI:10.1038/sj.onc.1202237. PMID:9872330.
• Li B، Tournier C، Davis RJ، Flavell RA (1999). "Regulation of IL-4 expression by the transcription factor JunB during T helper cell differentiation". EMBO J. ج. 18 ع. 2: 420–432. DOI:10.1093/emboj/18.2.420. PMC:1171136. PMID:9889198.
• Liberati NT، Datto MB، Frederick JP، Shen X، Wong C، Rougier-Chapman EM، Wang XF (1999). "Smads bind directly to the Jun family of AP-1 transcription factors". Proc. Natl. Acad. Sci. U.S.A. ج. 96 ع. 9: 4844–4849. DOI:10.1073/pnas.96.9.4844. PMC:21779. PMID:10220381.
• Chen P، Flory E، Avots A، Jordan BW، Kirchhoff F، Ludwig S، Rapp UR (2000). "Transactivation of naturally occurring HIV-1 long terminal repeats by the JNK signaling pathway. The most frequent naturally occurring length polymorphism sequence introduces a novel binding site for AP-1 factors". J. Biol. Chem. ج. 275 ع. 27: 20382–20390. DOI:10.1074/jbc.M001149200. PMID:10764760.
• Echlin DR، Tae HJ، Mitin N، Taparowsky EJ (2000). "B-ATF functions as a negative regulator of AP-1 mediated transcription and blocks cellular transformation by Ras and Fos". Oncogene. ج. 19 ع. 14: 1752–1763. DOI:10.1038/sj.onc.1203491. PMID:10777209.
• Verrecchia F، Pessah M، Atfi A، Mauviel A (2000). "Tumor necrosis factor-alpha inhibits transforming growth factor-beta /Smad signaling in human dermal fibroblasts via AP-1 activation". J. Biol. Chem. ج. 275 ع. 39: 30226–30231. DOI:10.1074/jbc.M005310200. PMID:10903323.