تضامنًا مع حق الشعب الفلسطيني |
FEZ1
An Error has occured retrieving Wikidata item for infobox FEZ1 (Fasciculation and elongation protein zeta 1) هوَ بروتين يُشَفر بواسطة جين FEZ1 في الإنسان.[1][2][3][3][4]
الوظيفة
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ Haedicke، J.؛ Brown، C.؛ Naghavi، M. H. (أغسطس 2009). "The brain-specific factor FEZ1 is a determinant of neuronal susceptibility to HIV-1 infection". Proceedings of the National Academy of Sciences. ج. 106 ع. 33: 14040–14045. Bibcode:2009PNAS..10614040H. DOI:10.1073/pnas.0900502106. ISSN:0027-8424. PMC:2729016. PMID:19667186.
- ^ Bloom L، Horvitz HR (مايو 1997). "The Caenorhabditis elegans gene unc-76 and its human homologs define a new gene family involved in axonal outgrowth and fasciculation". Proc Natl Acad Sci U S A. ج. 94 ع. 7: 3414–9. Bibcode:1997PNAS...94.3414B. DOI:10.1073/pnas.94.7.3414. PMC:20384. PMID:9096408.
- ^ أ ب "Entrez Gene: FEZ1 fasciculation and elongation protein zeta 1 (zygin I)". مؤرشف من الأصل في 2010-12-05.
- ^ Suzuki T، Okada Y، Semba S، Orba Y، Yamanouchi S، Endo S، Tanaka S، Fujita T، Kuroda S، Nagashima K، Sawa H (يونيو 2005). "Identification of FEZ1 as a protein that interacts with JC virus agnoprotein and microtubules: role of agnoprotein-induced dissociation of FEZ1 from microtubules in viral propagation". J Biol Chem. ج. 280 ع. 26: 24948–56. DOI:10.1074/jbc.M411499200. PMID:15843383.
قراءة متعمقة
- Kuroda S، Nakagawa N، Tokunaga C، وآخرون (1999). "Mammalian homologue of the Caenorhabditis elegans UNC-76 protein involved in axonal outgrowth is a protein kinase C zeta-interacting protein". J. Cell Biol. ج. 144 ع. 3: 403–11. DOI:10.1083/jcb.144.3.403. PMC:2132904. PMID:9971736.
- Whitehouse C، Chambers J، Howe K، وآخرون (2002). "NBR1 interacts with fasciculation and elongation protein zeta-1 (FEZ1) and calcium and integrin binding protein (CIB) and shows developmentally restricted expression in the neural tube". Eur. J. Biochem. ج. 269 ع. 2: 538–45. DOI:10.1046/j.0014-2956.2001.02681.x. PMID:11856312.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. Bibcode:2002PNAS...9916899M. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Miyoshi K، Honda A، Baba K، وآخرون (2004). "Disrupted-In-Schizophrenia 1, a candidate gene for schizophrenia, participates in neurite outgrowth". Mol. Psychiatry. ج. 8 ع. 7: 685–94. DOI:10.1038/sj.mp.4001352. PMID:12874605.
- Surpili MJ، Delben TM، Kobarg J (2004). "Identification of proteins that interact with the central coiled-coil region of the human protein kinase NEK1". Biochemistry. ج. 42 ع. 51: 15369–76. DOI:10.1021/bi034575v. PMID:14690447.
- Goehler H، Lalowski M، Stelzl U، وآخرون (2004). "A protein interaction network links GIT1, an enhancer of huntingtin aggregation, to Huntington's disease". Mol. Cell. ج. 15 ع. 6: 853–65. DOI:10.1016/j.molcel.2004.09.016. PMID:15383276.
- Okumura F، Hatakeyama S، Matsumoto M، وآخرون (2005). "Functional regulation of FEZ1 by the U-box-type ubiquitin ligase E4B contributes to neuritogenesis". J. Biol. Chem. ج. 279 ع. 51: 53533–43. DOI:10.1074/jbc.M402916200. PMID:15466860.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Yamada K، Nakamura K، Minabe Y، وآخرون (2005). "Association analysis of FEZ1 variants with schizophrenia in Japanese cohorts". Biol. Psychiatry. ج. 56 ع. 9: 683–90. DOI:10.1016/j.biopsych.2004.08.015. PMID:15522253.
- Stelzl U، Worm U، Lalowski M، وآخرون (2005). "A human protein-protein interaction network: a resource for annotating the proteome". Cell. ج. 122 ع. 6: 957–68. DOI:10.1016/j.cell.2005.08.029. PMID:16169070.
- Assmann EM، Alborghetti MR، Camargo ME، Kobarg J (2006). "FEZ1 dimerization and interaction with transcription regulatory proteins involves its coiled-coil region". J. Biol. Chem. ج. 281 ع. 15: 9869–81. DOI:10.1074/jbc.M513280200. PMID:16484223.
- Blasius TL، Cai D، Jih GT، وآخرون (2007). "Two binding partners cooperate to activate the molecular motor Kinesin-1". J. Cell Biol. ج. 176 ع. 1: 11–7. DOI:10.1083/jcb.200605099. PMC:2063617. PMID:17200414.
- Koga M، Ishiguro H، Horiuchi Y، وآخرون (2007). "Failure to confirm the association between the FEZ1 gene and schizophrenia in a Japanese population". Neurosci. Lett. ج. 417 ع. 3: 326–9. DOI:10.1016/j.neulet.2007.02.055. PMID:17374448.