APOBEC3F

An Error has occured retrieving Wikidata item for infobox APOBEC3F‏ (Apolipoprotein B mRNA editing enzyme catalytic subunit 3F) هوَ بروتين يُشَفر بواسطة جين APOBEC3F في الإنسان.^[1]^[2]^[3]

الوظيفة

الأهمية السريرية

المراجع

^ Holmes RK، Koning FA، Bishop KN، Malim MH (يناير 2007). "APOBEC3F can inhibit the accumulation of HIV-1 reverse transcription products in the absence of hypermutation. Comparisons with APOBEC3G". J Biol Chem. ج. 282 ع. 4: 2587–95. DOI:10.1074/jbc.M607298200. PMID:17121840.
^ "Entrez Gene: APOBEC3F apolipoprotein B mRNA editing enzyme, catalytic polypeptide-like 3F". مؤرشف من الأصل في 2010-12-05.
^ Jarmuz A، Chester A، Bayliss J، Gisbourne J، Dunham I، Scott J، Navaratnam N (فبراير 2002). "An anthropoid-specific locus of orphan C to U RNA-editing enzymes on chromosome 22". Genomics. ج. 79 ع. 3: 285–96. DOI:10.1006/geno.2002.6718. PMID:11863358.

قراءة متعمقة

Wedekind JE، Dance GS، Sowden MP، Smith HC (2003). "Messenger RNA editing in mammals: new members of the APOBEC family seeking roles in the family business". Trends Genet. ج. 19 ع. 4: 207–16. DOI:10.1016/S0168-9525(03)00054-4. PMID:12683974.
Franca R، Spadari S، Maga G (2006). "APOBEC deaminases as cellular antiviral factors: a novel natural host defense mechanism". Med. Sci. Monit. ج. 12 ع. 5: RA92–8. PMID:16641889.
Prashar Y، Weissman SM (1996). "Analysis of differential gene expression by display of 3' end restriction fragments of cDNAs". Proc. Natl. Acad. Sci. U.S.A. ج. 93 ع. 2: 659–63. DOI:10.1073/pnas.93.2.659. PMC:40108. PMID:8570611.
Bonaldo MF، Lennon G، Soares MB (1997). "Normalization and subtraction: two approaches to facilitate gene discovery". Genome Res. ج. 6 ع. 9: 791–806. DOI:10.1101/gr.6.9.791. PMID:8889548.
Dunham I، Shimizu N، Roe BA، وآخرون (1999). "The DNA sequence of human chromosome 22". Nature. ج. 402 ع. 6761: 489–95. DOI:10.1038/990031. PMID:10591208.
Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
Zheng YH، Irwin D، Kurosu T، وآخرون (2004). "Human APOBEC3F is another host factor that blocks human immunodeficiency virus type 1 replication". J. Virol. ج. 78 ع. 11: 6073–6. DOI:10.1128/JVI.78.11.6073-6076.2004. PMC:415831. PMID:15141007.
Wiegand HL، Doehle BP، Bogerd HP، Cullen BR (2004). "A second human antiretroviral factor, APOBEC3F, is suppressed by the HIV-1 and HIV-2 Vif proteins". EMBO J. ج. 23 ع. 12: 2451–8. DOI:10.1038/sj.emboj.7600246. PMC:423288. PMID:15152192.
Liddament MT، Brown WL، Schumacher AJ، Harris RS (2004). "APOBEC3F properties and hypermutation preferences indicate activity against HIV-1 in vivo". Curr. Biol. ج. 14 ع. 15: 1385–91. DOI:10.1016/j.cub.2004.06.050. PMID:15296757.
Collins JE، Wright CL، Edwards CA، وآخرون (2005). "A genome annotation-driven approach to cloning the human ORFeome". Genome Biol. ج. 5 ع. 10: R84. DOI:10.1186/gb-2004-5-10-r84. PMC:545604. PMID:15461802.
Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
Haché G، Liddament MT، Harris RS (2005). "The retroviral hypermutation specificity of APOBEC3F and APOBEC3G is governed by the C-terminal DNA cytosine deaminase domain". J. Biol. Chem. ج. 280 ع. 12: 10920–4. DOI:10.1074/jbc.M500382200. PMID:15647250.
Kimura K، Wakamatsu A، Suzuki Y، وآخرون (2006). "Diversification of transcriptional modulation: large-scale identification and characterization of putative alternative promoters of human genes". Genome Res. ج. 16 ع. 1: 55–65. DOI:10.1101/gr.4039406. PMC:1356129. PMID:16344560.
Zennou V، Bieniasz PD (2006). "Comparative analysis of the antiretroviral activity of APOBEC3G and APOBEC3F from primates". Virology. ج. 349 ع. 1: 31–40. DOI:10.1016/j.virol.2005.12.035. PMID:16460778.
Tian C، Yu X، Zhang W، وآخرون (2006). "Differential requirement for conserved tryptophans in human immunodeficiency virus type 1 Vif for the selective suppression of APOBEC3G and APOBEC3F". J. Virol. ج. 80 ع. 6: 3112–5. DOI:10.1128/JVI.80.6.3112-3115.2006. PMC:1395459. PMID:16501124.
Stenglein MD، Harris RS (2006). "APOBEC3B and APOBEC3F inhibit L1 retrotransposition by a DNA deamination-independent mechanism". J. Biol. Chem. ج. 281 ع. 25: 16837–41. DOI:10.1074/jbc.M602367200. PMID:16648136.
Wichroski MJ، Robb GB، Rana TM (2006). "Human retroviral host restriction factors APOBEC3G and APOBEC3F localize to mRNA processing bodies". PLoS Pathog. ج. 2 ع. 5: e41. DOI:10.1371/journal.ppat.0020041. PMC:1458959. PMID:16699599.

هذه بذرة مقالة عن بروتين بحاجة للتوسيع. فضلًا شارك في تحريرها.

[1] Holmes RK، Koning FA، Bishop KN، Malim MH (يناير 2007). "APOBEC3F can inhibit the accumulation of HIV-1 reverse transcription products in the absence of hypermutation. Comparisons with APOBEC3G". J Biol Chem. ج. 282 ع. 4: 2587–95. DOI:10.1074/jbc.M607298200. PMID:17121840.

[2] "Entrez Gene: APOBEC3F apolipoprotein B mRNA editing enzyme, catalytic polypeptide-like 3F". مؤرشف من الأصل في 2010-12-05.

[3] Jarmuz A، Chester A، Bayliss J، Gisbourne J، Dunham I، Scott J، Navaratnam N (فبراير 2002). "An anthropoid-specific locus of orphan C to U RNA-editing enzymes on chromosome 22". Genomics. ج. 79 ع. 3: 285–96. DOI:10.1006/geno.2002.6718. PMID:11863358.

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