An Error has occured retrieving Wikidata item for infobox TPD52L2‏ (TPD52L2 protein) هوَ بروتين يُشَفر بواسطة جين TPD52L2 في الإنسان.[1][2]

الوظيفة

الأهمية السريرية

المراجع

  1. ^ "Entrez Gene: TPD52L2 tumor protein D52-like 2". مؤرشف من الأصل في 2010-12-05.
  2. ^ Byrne JA، Nourse CR، Basset P، Gunning P (مارس 1998). "Identification of homo- and heteromeric interactions between members of the breast carcinoma-associated D52 protein family using the yeast two-hybrid system". Oncogene. ج. 16 ع. 7: 873–81. DOI:10.1038/sj.onc.1201604. PMID:9484778.

قراءة متعمقة

  • Maruyama K، Sugano S (1994). "Oligo-capping: a simple method to replace the cap structure of eukaryotic mRNAs with oligoribonucleotides". Gene. ج. 138 ع. 1–2: 171–4. DOI:10.1016/0378-1119(94)90802-8. PMID:8125298.
  • Byrne JA، Mattei MG، Basset P (1996). "Definition of the tumor protein D52 (TPD52) gene family through cloning of D52 homologues in human (hD53) and mouse (mD52)". Genomics. ج. 35 ع. 3: 523–32. DOI:10.1006/geno.1996.0393. PMID:8812487.
  • Suzuki Y، Yoshitomo-Nakagawa K، Maruyama K، Suyama A، Sugano S (1997). "Construction and characterization of a full length-enriched and a 5'-end-enriched cDNA library". Gene. ج. 200 ع. 1–2: 149–56. DOI:10.1016/S0378-1119(97)00411-3. PMID:9373149.
  • Nourse CR، Mattei MG، Gunning P، Byrne JA (1998). "Cloning of a third member of the D52 gene family indicates alternative coding sequence usage in D52-like transcripts". Biochim. Biophys. Acta. ج. 1443 ع. 1–2: 155–68. DOI:10.1016/S0167-4781(98)00211-5. PMID:9838088.
  • Wilson SH، Bailey AM، Nourse CR، Mattei MG، Byrne JA (2001). "Identification of MAL2, a novel member of the mal proteolipid family, though interactions with TPD52-like proteins in the yeast two-hybrid system". Genomics. ج. 76 ع. 1–3: 81–8. DOI:10.1006/geno.2001.6610. PMID:11549320.
  • Boutros R، Byrne JA (2005). "D53 (TPD52L1) is a cell cycle-regulated protein maximally expressed at the G2-M transition in breast cancer cells". Exp. Cell Res. ج. 310 ع. 1: 152–65. DOI:10.1016/j.yexcr.2005.07.009. PMID:16112108.
  • Kimura K، Wakamatsu A، Suzuki Y، Ota T، Nishikawa T، Yamashita R، Yamamoto J، Sekine M، Tsuritani K، Wakaguri H، Ishii S، Sugiyama T، Saito K، Isono Y، Irie R، Kushida N، Yoneyama T، Otsuka R، Kanda K، Yokoi T، Kondo H، Wagatsuma M، Murakawa K، Ishida S، Ishibashi T، Takahashi-Fujii A، Tanase T، Nagai K، Kikuchi H، Nakai K، Isogai T، Sugano S (2006). "Diversification of transcriptional modulation: large-scale identification and characterization of putative alternative promoters of human genes". Genome Res. ج. 16 ع. 1: 55–65. DOI:10.1101/gr.4039406. PMC:1356129. PMID:16344560.
  • Barbaric D، Byth K، Dalla-Pozza L، Byrne JA (2006). "Expression of tumor protein D52-like genes in childhood leukemia at diagnosis: clinical and sample considerations". Leuk. Res. ج. 30 ع. 11: 1355–63. DOI:10.1016/j.leukres.2006.03.009. PMID:16620967.
  • Cao Q، Chen J، Zhu L، Liu Y، Zhou Z، Sha J، Wang S، Li J (2006). "A testis-specific and testis developmentally regulated tumor protein D52 (TPD52)-like protein TPD52L3/hD55 interacts with TPD52 family proteins". Biochem. Biophys. Res. Commun. ج. 344 ع. 3: 798–806. DOI:10.1016/j.bbrc.2006.03.208. PMID:16631610.
  • Olsen JV، Blagoev B، Gnad F، Macek B، Kumar C، Mortensen P، Mann M (2006). "Global, in vivo, and site-specific phosphorylation dynamics in signaling networks". Cell. ج. 127 ع. 3: 635–48. DOI:10.1016/j.cell.2006.09.026. PMID:17081983.