SLAMF6
An Error has occured retrieving Wikidata item for infobox SLAMF6 (SLAM family member 6) هوَ بروتين يُشَفر بواسطة جين SLAMF6 في الإنسان.[1][2]
الوظيفة
![[أيقونة]](/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ "Entrez Gene: SLAMF6 SLAM family member 6". مؤرشف من الأصل في 2010-12-05.
- ^ Bottino C، Falco M، Parolini S، Marcenaro E، Augugliaro R، Sivori S، Landi E، Biassoni R، Notarangelo LD، Moretta L، Moretta A (أغسطس 2001). "NTB-A [correction of GNTB-A], a novel SH2D1A-associated surface molecule contributing to the inability of natural killer cells to kill Epstein-Barr virus-infected B cells in X-linked lymphoproliferative disease". J Exp Med. ج. 194 ع. 3: 235–46. DOI:10.1084/jem.194.3.235. PMC:2193462. PMID:11489943.
قراءة متعمقة
- Claus M، Meinke S، Bhat R، Watzl C (2007). "Regulation of NK cell activity by 2B4, NTB-A and CRACC". Front. Biosci. ج. 13 ع. 13: 956–65. DOI:10.2741/2735. PMID:17981603.
- Hercend T، Meuer S، Brennan A، وآخرون (1984). "Natural killer-like function of activated T lymphocytes: differential blocking effects of monoclonal antibodies specific for a 90-kDa clonotypic structure". Cell. Immunol. ج. 86 ع. 2: 381–92. DOI:10.1016/0008-8749(84)90393-9. PMID:6610481.
- Kong G، Dalton M، Wardenburg JB، وآخرون (1996). "Distinct tyrosine phosphorylation sites in ZAP-70 mediate activation and negative regulation of antigen receptor function". Mol. Cell. Biol. ج. 16 ع. 9: 5026–35. PMC:231504. PMID:8756661.
- Lee YJ، Luisiri P، Clark MR (1996). "A novel complex, p40/42, is constitutively associated with the B cell antigen receptor and phosphorylated upon receptor stimulation". J. Immunol. ج. 157 ع. 9: 3828–37. PMID:8892612.
- Gray CW، Ward RV، Karran E، وآخرون (2000). "Characterization of human HtrA2, a novel serine protease involved in the mammalian cellular stress response". Eur. J. Biochem. ج. 267 ع. 18: 5699–710. DOI:10.1046/j.1432-1327.2000.01589.x. PMID:10971580.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Del Valle JM، Engel P، Martín M (2003). "The cell surface expression of SAP-binding receptor CD229 is regulated via its interaction with clathrin-associated adaptor complex 2 (AP-2)". J. Biol. Chem. ج. 278 ع. 19: 17430–7. DOI:10.1074/jbc.M301569200. PMID:12621057.
- Tangye SG، Nichols KE، Hare NJ، van de Weerdt BC (2003). "Functional requirements for interactions between CD84 and Src homology 2 domain-containing proteins and their contribution to human T cell activation". J. Immunol. ج. 171 ع. 5: 2485–95. DOI:10.4049/jimmunol.171.5.2485. PMID:12928397.
- Clark HF، Gurney AL، Abaya E، وآخرون (2003). "The secreted protein discovery initiative (SPDI), a large-scale effort to identify novel human secreted and transmembrane proteins: a bioinformatics assessment". Genome Res. ج. 13 ع. 10: 2265–70. DOI:10.1101/gr.1293003. PMC:403697. PMID:12975309.
- Valdez PA، Wang H، Seshasayee D، وآخرون (2004). "NTB-A, a new activating receptor in T cells that regulates autoimmune disease". J. Biol. Chem. ج. 279 ع. 18: 18662–9. DOI:10.1074/jbc.M312313200. PMID:14988414.
- Flaig RM، Stark S، Watzl C (2004). "Cutting edge: NTB-A activates NK cells via homophilic interaction". J. Immunol. ج. 172 ع. 11: 6524–7. DOI:10.4049/jimmunol.172.11.6524. PMID:15153464.
- Zhang Z، Henzel WJ (2005). "Signal peptide prediction based on analysis of experimentally verified cleavage sites". Protein Sci. ج. 13 ع. 10: 2819–24. DOI:10.1110/ps.04682504. PMC:2286551. PMID:15340161.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Stark S، Watzl C (2006). "2B4 (CD244), NTB-A and CRACC (CS1) stimulate cytotoxicity but no proliferation in human NK cells". Int. Immunol. ج. 18 ع. 2: 241–7. DOI:10.1093/intimm/dxh358. PMID:16410313.
- Gregory SG، Barlow KF، McLay KE، وآخرون (2006). "The DNA sequence and biological annotation of human chromosome 1". Nature. ج. 441 ع. 7091: 315–21. DOI:10.1038/nature04727. PMID:16710414.
- Eissmann P، Watzl C (2006). "Molecular analysis of NTB-A signaling: a role for EAT-2 in NTB-A-mediated activation of human NK cells". J. Immunol. ج. 177 ع. 5: 3170–7. DOI:10.4049/jimmunol.177.5.3170. PMID:16920955.
- Cao E، Ramagopal UA، Fedorov A، وآخرون (2006). "NTB-A receptor crystal structure: insights into homophilic interactions in the signaling lymphocytic activation molecule receptor family". Immunity. ج. 25 ع. 4: 559–70. DOI:10.1016/j.immuni.2006.06.020. PMID:17045824.
- Banerjee P، Feuer G، Barker E (2007). "Human T-cell leukemia virus type 1 (HTLV-1) p12I down-modulates ICAM-1 and -2 and reduces adherence of natural killer cells, thereby protecting HTLV-1-infected primary CD4+ T cells from autologous natural killer cell-mediated cytotoxicity despite the reduction of major histocompatibility complex class I molecules on infected cells". J. Virol. ج. 81 ع. 18: 9707–17. DOI:10.1128/JVI.00887-07. PMC:2045425. PMID:17609265.
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