PKIA
An Error has occured retrieving Wikidata item for infobox PKIA (CAMP-dependent protein kinase inhibitor alpha) هوَ بروتين يُشَفر بواسطة جين PKIA في الإنسان.[1][2]
الوظيفة
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ Olsen SR، Uhler MD (فبراير 1992). "Inhibition of protein kinase-A by overexpression of the cloned human protein kinase inhibitor". Mol Endocrinol. ج. 5 ع. 9: 1246–56. DOI:10.1210/mend-5-9-1246. PMID:1770951.
- ^ "Entrez Gene: PKIA protein kinase (cAMP-dependent, catalytic) inhibitor alpha". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Olsen SR، Uhler MD (1991). "Isolation and characterization of cDNA clones for an inhibitor protein of cAMP-dependent protein kinase". J. Biol. Chem. ج. 266 ع. 17: 11158–62. PMID:1710219.
- Knighton DR، Zheng JH، Ten Eyck LF، وآخرون (1991). "Structure of a peptide inhibitor bound to the catalytic subunit of cyclic adenosine monophosphate-dependent protein kinase". Science. ج. 253 ع. 5018: 414–20. DOI:10.1126/science.1862343. PMID:1862343.
- Van Patten SM، Heisermann GJ، Cheng HC، Walsh DA (1987). "Tyrosine kinase catalyzed phosphorylation and inactivation of the inhibitor protein of the cAMP-dependent protein kinase". J. Biol. Chem. ج. 262 ع. 7: 3398–403. PMID:2434500.
- Scott JD، Fischer EH، Demaille JG، Krebs EG (1985). "Identification of an inhibitory region of the heat-stable protein inhibitor of the cAMP-dependent protein kinase". Proc. Natl. Acad. Sci. U.S.A. ج. 82 ع. 13: 4379–83. DOI:10.1073/pnas.82.13.4379. PMC:390417. PMID:2989819.
- Scott JD، Glaccum MB، Fischer EH، Krebs EG (1986). "Primary-structure requirements for inhibition by the heat-stable inhibitor of the cAMP-dependent protein kinase". Proc. Natl. Acad. Sci. U.S.A. ج. 83 ع. 6: 1613–6. DOI:10.1073/pnas.83.6.1613. PMC:323133. PMID:3456605.
- Wen W، Taylor SS، Meinkoth JL (1995). "The expression and intracellular distribution of the heat-stable protein kinase inhibitor is cell cycle regulated". J. Biol. Chem. ج. 270 ع. 5: 2041–6. DOI:10.1074/jbc.270.5.2041. PMID:7836431.
- Baude EJ، Dignam SS، Olsen SR، وآخرون (1994). "Glutamic acid 203 of the cAMP-dependent protein kinase catalytic subunit participates in the inhibition by two isoforms of the protein kinase inhibitor". J. Biol. Chem. ج. 269 ع. 3: 2316–23. PMID:7905001.
- Baude EJ، Dignam SS، Reimann EM، Uhler MD (1994). "Evidence for the importance of hydrophobic residues in the interactions between the cAMP-dependent protein kinase catalytic subunit and the protein kinase inhibitors". J. Biol. Chem. ج. 269 ع. 27: 18128–33. PMID:8027074.
- Fritz CC، Green MR (1997). "HIV Rev uses a conserved cellular protein export pathway for the nucleocytoplasmic transport of viral RNAs". Curr. Biol. ج. 6 ع. 7: 848–54. DOI:10.1016/S0960-9822(02)00608-5. PMID:8805303.
- Zimmermann B، Chiorini JA، Ma Y، وآخرون (1999). "PrKX is a novel catalytic subunit of the cAMP-dependent protein kinase regulated by the regulatory subunit type I". J. Biol. Chem. ج. 274 ع. 9: 5370–8. DOI:10.1074/jbc.274.9.5370. PMID:10026146.
- Yu J، Yu L، Chen Z، وآخرون (2002). "Protein inhibitor of neuronal nitric oxide synthase interacts with protein kinase A inhibitors". Brain Res. Mol. Brain Res. ج. 99 ع. 2: 145–9. DOI:10.1016/S0169-328X(02)00104-3. PMID:11978406.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Gassel M، Breitenlechner CB، Rüger P، وآخرون (2003). "Mutants of protein kinase A that mimic the ATP-binding site of protein kinase B (AKT)". J. Mol. Biol. ج. 329 ع. 5: 1021–34. DOI:10.1016/S0022-2836(03)00518-7. PMID:12798691.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.