CLCF1
An Error has occured retrieving Wikidata item for infobox CLCF1 (Cardiotrophin like cytokine factor 1) هوَ بروتين يُشَفر بواسطة جين CLCF1 في الإنسان.[1]
الوظيفة
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ "Entrez Gene: CLCF1 cardiotrophin-like cytokine factor 1". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Shi Y، Wang W، Yourey PA، وآخرون (1999). "Computational EST database analysis identifies a novel member of the neuropoietic cytokine family". Biochem. Biophys. Res. Commun. ج. 262 ع. 1: 132–8. DOI:10.1006/bbrc.1999.1181. PMID:10448081.
- Senaldi G، Varnum BC، Sarmiento U، وآخرون (1999). "Novel neurotrophin-1/B cell-stimulating factor-3: a cytokine of the IL-6 family". Proc. Natl. Acad. Sci. U.S.A. ج. 96 ع. 20: 11458–63. DOI:10.1073/pnas.96.20.11458. PMC:18055. PMID:10500198.
- Elson GC، Lelièvre E، Guillet C، وآخرون (2000). "CLF associates with CLC to form a functional heteromeric ligand for the CNTF receptor complex". Nat. Neurosci. ج. 3 ع. 9: 867–72. DOI:10.1038/78765. PMID:10966616.
- Plun-Favreau H، Elson G، Chabbert M، وآخرون (2001). "The ciliary neurotrophic factor receptor alpha component induces the secretion of and is required for functional responses to cardiotrophin-like cytokine". EMBO J. ج. 20 ع. 7: 1692–703. DOI:10.1093/emboj/20.7.1692. PMC:145510. PMID:11285233.
- Lelièvre E، Plun-Favreau H، Chevalier S، وآخرون (2001). "Signaling pathways recruited by the cardiotrophin-like cytokine/cytokine-like factor-1 composite cytokine: specific requirement of the membrane-bound form of ciliary neurotrophic factor receptor alpha component". J. Biol. Chem. ج. 276 ع. 25: 22476–84. DOI:10.1074/jbc.M101681200. PMID:11294841.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Auernhammer CJ، Isele NB، Kopp FB، وآخرون (2003). "Novel neurotrophin-1/B cell-stimulating factor-3 (cardiotrophin-like cytokine) stimulates corticotroph function via a signal transducer and activator of transcription-dependent mechanism negatively regulated by suppressor of cytokine signaling-3". Endocrinology. ج. 144 ع. 4: 1202–10. DOI:10.1210/en.2002-220933. PMID:12639901.
- Burger R، Bakker F، Guenther A، وآخرون (2004). "Functional significance of novel neurotrophin-1/B cell-stimulating factor-3 (cardiotrophin-like cytokine) for human myeloma cell growth and survival". Br. J. Haematol. ج. 123 ع. 5: 869–78. DOI:10.1046/j.1365-2141.2003.04686.x. PMID:14632778.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Rousseau F، Gauchat JF، McLeod JG، وآخرون (2006). "Inactivation of cardiotrophin-like cytokine, a second ligand for ciliary neurotrophic factor receptor, leads to cold-induced sweating syndrome in a patient". Proc. Natl. Acad. Sci. U.S.A. ج. 103 ع. 26: 10068–73. DOI:10.1073/pnas.0509598103. PMC:1502507. PMID:16782820.
- Dagoneau N، Bellais S، Blanchet P، وآخرون (2007). "Mutations in cytokine receptor-like factor 1 (CRLF1) account for both Crisponi and cold-induced sweating syndromes". Am. J. Hum. Genet. ج. 80 ع. 5: 966–70. DOI:10.1086/513608. PMC:1852726. PMID:17436251.